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Nerve growth factor signaling, neuroprotection, and neural repair
Hemoglobin and the paracrine and endocrine functions of nitric oxide
In addition, this parameter was recorded for the study of lipid profile, considering the demonstrated role of oxidative stress, both in the induction of cardiac arrhythmias and in lipid oxidation with subsequent development of an immune response, with a pathogenic evolution towards accelerated atherosclerotic endothelial dysfunction (4)
Glutathione (GSH) levels were considerably reduced, increasing the ROS/GSH ratio, significantly decreasing the expression of the autophagy protein LC3B and related genes, and substantially increasing the expression of the caspase3 protein and apoptosis-related genes in apoptosis assays
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